Nejdále sedlo zpětná vazba pa tu 8902 zachránit Potlačení oholit se
Identification of antibody reactive proteins in pancreatic cancer using 2D immunoblotting and mass spectrometry
Leibniz Institute | PA-TU-8902 | DSMZ
Expression of MRP4 and MRP5 in pancreatic cancer-derived cell lines. A,... | Download Scientific Diagram
PaTu 8902 Pharmacogenomics Data | CellMinerCDB | NCI Genomics and Pharmacology Facility
Identification of antibody reactive proteins in pancreatic cancer using 2D immunoblotting and mass spectrometry
Cell division rate and MTT viability measurement in PaTu 8902 cells... | Download Scientific Diagram
PDA requires GOT1 for cellular proliferation and tumor growth. a... | Download Scientific Diagram
Heme oxygenase is not involved in the anti-proliferative effects of statins on pancreatic cancer cells | BMC Cancer | Full Text
Cancers | Free Full-Text | Effective Oncoleaking Treatment of Pancreatic Cancer by Claudin-Targeted Suicide Gene Therapy with Clostridium perfringens Enterotoxin (CPE)
Desmoplastic Crosstalk in Pancreatic Ductal Adenocarcinoma Is Reflected by Different Responses of Panc-1, MIAPaCa-2, PaTu-8902, and CAPAN-2 Cell Lines to Cancer-associated/Normal Fibroblasts | Cancer Genomics & Proteomics
The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2 | bioRxiv
Heme oxygenase is not involved in the anti-proliferative effects of statins on pancreatic cancer cells | BMC Cancer | Full Text
Leibniz Institute | PA-TU-8902 | DSMZ
German Collection of Microorganisms and Cell Cultures GmbH: Details
The Casp3/7 activity in control cells. The luminescence readout of... | Download Scientific Diagram
Pancreatic cancer cell/fibroblast co-culture induces M2 like macrophages that influence therapeutic response in a 3D model | PLOS ONE
SMAD4 loss limits the vulnerability of pancreatic cancer cells to complex I inhibition via promotion of mitophagy | Oncogene
The details of time-lapse microscopy figures showing control PaTu 8902... | Download Scientific Diagram
The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2 | bioRxiv
Desmoplastic Crosstalk in Pancreatic Ductal Adenocarcinoma Is Reflected by Different Responses of Panc-1, MIAPaCa-2, PaTu-8902, and CAPAN-2 Cell Lines to Cancer-associated/Normal Fibroblasts | Cancer Genomics & Proteomics
German Collection of Microorganisms and Cell Cultures GmbH: Details
Comparison of Oct4, Sox2 and Nanog Expression in Pancreatic Cancer Cell Lines and Human Pancreatic Tumor | Zahedan Journal of Research in Medical Sciences | Full Text
The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2 | bioRxiv
Cancers | Free Full-Text | Effective Oncoleaking Treatment of Pancreatic Cancer by Claudin-Targeted Suicide Gene Therapy with Clostridium perfringens Enterotoxin (CPE)
GOT1 inhibition promotes pancreatic cancer cell death by ferroptosis | Nature Communications
The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2 | bioRxiv
IJMS | Free Full-Text | Inhibition of Mitochondrial Metabolism Leads to Selective Eradication of Cells Adapted to Acidic Microenvironment
Pancreatic cancer cell/fibroblast co-culture induces M2 like macrophages that influence therapeutic response in a 3D model | PLOS ONE